{"id":2012,"date":"2014-10-01T01:00:00","date_gmt":"2014-10-01T00:00:00","guid":{"rendered":"https:\/\/angh.net\/abstracts\/impact-des-traitements-anticoagulants-et-anti-agregants-plaquettaires-sur-la-morbi-mortalite-des-hemorragies-de-lhypertension-portale-sur-cirrhose\/"},"modified":"2018-10-20T18:40:15","modified_gmt":"2018-10-20T16:40:15","slug":"impact-des-traitements-anticoagulants-et-anti-agregants-plaquettaires-sur-la-morbi-mortalite-des-hemorragies-de-lhypertension-portale-sur-cirrhose","status":"publish","type":"post","link":"https:\/\/angh.net\/abstracts\/impact-des-traitements-anticoagulants-et-anti-agregants-plaquettaires-sur-la-morbi-mortalite-des-hemorragies-de-lhypertension-portale-sur-cirrhose\/","title":{"rendered":"Impact des traitements anticoagulants et anti-agr\u00e9gants plaquettaires sur la morbi-mortalit\u00e9 des h\u00e9morragies de l\u2019hypertension portale sur cirrhose"},"content":{"rendered":"<p><strong>2014<\/strong><\/p>\n<p><em>Le Bricquir Y (B\u00e9ziers), R\u00e9my AJ (Perpignan), Cadranel JF (Creil), Causse X (Orl\u00e9ans), Dadamessi I (Saint-Quentin), Aziz K (Saint-Brieuc), Elriz K (Evry-Corbeil), Pofelski J (Annecy), Bellaiche G (Aulnay s\/Bois), Bramli S (Avignon), Lamare L (Lorient), Ah-Soune P (Toulon), Skinazi F (Saint-Denis), Dupuychaffray JP (Angoul\u00eame), Pariente A (Pau), Henrion J (Jolimont), Vitte RL (Poissy), Bour B (Le Mans), Seyrig JA (Pontivy), Dewaele F (La Roche-sur-Yon), Macaigne G (Lagny), Zerouala F (Meaux), de Montigny S (Aubagne), Payen JL (Montauban), Jouannaud V (Monfermeil), Doumet S (Villeneuve Saint-Georges), Guivarch P (Castres), Donato L (Vernon), D&rsquo;Harondel C (Provins), P\u00e9laquier A (Mont\u00e9limar), Pauwels A (Gonesse), pour l\u2019ANGH ; Thabut D, pour le CFHTP<\/em><\/p>\n<p><strong>H\u00e9patologie <\/strong>&#8211; \u00a02014-05-11 &#8211;\u00a0CO &#8211;<\/p>\n<p>________________________________<\/p>\n<p>Introduction : La cirrhose est parfois associ\u00e9e \u00e0 des pathologies (thrombose portale, ACFA, maladies vasculaires isch\u00e9miques) n\u00e9cessitant un traitement anticoagulant ou anti-agr\u00e9gant plaquettaire. Cependant, l\u2019une de ses principales complications est l\u2019h\u00e9morragie digestive li\u00e9e \u00e0 l\u2019hypertension portale (HTP). L\u2019objectif de ce travail \u00e9tait d\u2019\u00e9valuer l\u2019impact des anticoagulants et des anti-agr\u00e9gants plaquettaires sur la s\u00e9v\u00e9rit\u00e9 et l\u2019\u00e9volution des h\u00e9morragies de l\u2019HTP sur cirrhose.<br \/>\nM\u00e9thodes : 57 centres (26 CHU, 31 CHG) ont particip\u00e9 (mars 2012-avril 2013) \u00e0 une \u00e9tude observationnelle prospective sur les h\u00e9morragies de l\u2019HTP sur cirrhose (observatoire CHOC). 891 patients ont \u00e9t\u00e9 inclus. 147 (16,6%) recevaient un traitement anticoagulant et\/ou anti-agr\u00e9gant plaquettaire. Les patients ont \u00e9t\u00e9 r\u00e9partis en 4 groupes : anticoagulant (gr. 1, n=55), anti-agr\u00e9gant (gr. 2, n=83), anticoagulant+anti-agr\u00e9gant (gr. 3, n=9), pas d\u2019anticoagulant\/anti-agr\u00e9gant (gr. 4).<br \/>\nR\u00e9sultats : Les patients du groupe 1 (anticoagulant) \u00e9taient plus \u00e2g\u00e9s (66 vs 58 ans, p&lt;0,0001) et avaient une cr\u00e9atinin\u00e9mie plus \u00e9lev\u00e9e (146 vs 90 \u00b5mol\/l, p&lt;0,0001) que les patients du groupe 4, mais ne diff\u00e9raient pas d\u2019eux pour les param\u00e8tres de fonction h\u00e9patique, hormis l\u2019INR (2,63 vs 1,96, p&lt;0,004). En termes de s\u00e9v\u00e9rit\u00e9 et d\u2019\u00e9volution des h\u00e9morragies de l\u2019HTP, aucune diff\u00e9rence significative n\u2019\u00e9tait observ\u00e9e entre les deux groupes.<br \/>\nLes patients du groupe 2 (anti-agr\u00e9gant) \u00e9taient plus \u00e2g\u00e9s (68 vs 58 ans, p&lt;0,0001) et avaient une fonction h\u00e9patique moins alt\u00e9r\u00e9e (score de Child : 7,9 vs 9, p&lt;0,001) que ceux du groupe 4. Chez les patients Child A et B, il n\u2019y avait pas de diff\u00e9rence significative entre les deux groupes en termes de s\u00e9v\u00e9rit\u00e9 (choc : 16 vs 13%, saignement actif \u00e0 l\u2019endoscopie : 35 vs 34%, patients transfus\u00e9s : 73 vs 66%) et d\u2019\u00e9volution (non-contr\u00f4le de l\u2019h\u00e9morragie : 5,3 vs 5%, mortalit\u00e9 J42 : 11,6 vs 8,6%) de l\u2019h\u00e9morragie. En revanche, chez les patients Child C, un saignement actif \u00e0 l\u2019endoscopie (64 vs 42%) et le non-contr\u00f4le de l\u2019h\u00e9morragie (29 vs 11%) tendaient \u00e0 \u00eatre plus fr\u00e9quents, tandis que la mortalit\u00e9 \u00e0 J42 (50 vs 37%, p&lt;0,03) \u00e9tait significativement plus \u00e9lev\u00e9e, dans le groupe 2.<br \/>\nConclusion : Dans cette cohorte, 1) les anticoagulants n\u2019\u00e9taient pas associ\u00e9s \u00e0 une s\u00e9v\u00e9rit\u00e9 accrue des h\u00e9morragies de l\u2019HTP ; 2) les anti-agr\u00e9gants augmentaient la morbi-mortalit\u00e9 des h\u00e9morragies de l\u2019HTP chez les patients Child C ; \u00e0 l\u2019inverse, ils n\u2019avaient pas d\u2019impact significatif chez les patients Child A et B.<\/p>\n","protected":false},"excerpt":{"rendered":"<p>2014 Le Bricquir Y (B\u00e9ziers), R\u00e9my AJ (Perpignan), Cadranel JF (Creil), Causse X (Orl\u00e9ans), Dadamessi I (Saint-Quentin), Aziz K (Saint-Brieuc), Elriz K (Evry-Corbeil), Pofelski J (Annecy), Bellaiche G (Aulnay s\/Bois), Bramli S (Avignon), Lamare L (Lorient), Ah-Soune P (Toulon), Skinazi F (Saint-Denis), Dupuychaffray JP (Angoul\u00eame), Pariente A (Pau), Henrion J (Jolimont), Vitte RL (Poissy), Bour [&hellip;]<\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_exactmetrics_skip_tracking":false,"_exactmetrics_sitenote_active":false,"_exactmetrics_sitenote_note":"","_exactmetrics_sitenote_category":0,"footnotes":""},"categories":[3],"tags":[25],"class_list":["post-2012","post","type-post","status-publish","format-standard","hentry","category-hepatologie","tag-25"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.2 - 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