{"id":2054,"date":"2016-10-01T01:00:00","date_gmt":"2016-10-01T00:00:00","guid":{"rendered":"https:\/\/angh.net\/abstracts\/role-du-virus-de-lhepatite-e-dans-la-decompensation-de-la-cirrhose-alcoolique-donnees-dune-etude-prospective-et-multicentrique\/"},"modified":"2018-10-20T18:40:22","modified_gmt":"2018-10-20T16:40:22","slug":"role-du-virus-de-lhepatite-e-dans-la-decompensation-de-la-cirrhose-alcoolique-donnees-dune-etude-prospective-et-multicentrique","status":"publish","type":"post","link":"https:\/\/angh.net\/abstracts\/role-du-virus-de-lhepatite-e-dans-la-decompensation-de-la-cirrhose-alcoolique-donnees-dune-etude-prospective-et-multicentrique\/","title":{"rendered":"R\u00f4le du virus de l\u2019h\u00e9patite E dans la d\u00e9compensation de la cirrhose alcoolique\u00a0? Donn\u00e9es d\u2019une \u00e9tude prospective et multicentrique"},"content":{"rendered":"<p><strong>2016<\/strong><\/p>\n<p><em>C. Renou (CH Hy\u00e8res), B. Lesgourgues (CH Montfermeil), H. Agostini (H\u00f4pital Le Kremlin Bic\u00eatre, Le Kremlin Bic\u00eatre), G. Macaigne (CH Marne la Vall\u00e9e), A. Pauwels (CH Gonesse), Y. Le Bricquir (CH B\u00e9ziers), J. Henrion (CH Jolimont, Belgique), F. Khemissa (CH Perpignan), E. Clair (CH Montfermeil), T. Paupard (CH Dunkerque), A. Pelaquier (CH Mont\u00e9limar), H. Minazzi (CH Chamb\u00e9ry), S. Doumet (CH Villeneuve-Saint-Georges), A.Raoto (CH Avignon), C. Locher (CH Meaux), J.A. Seyrig (CH Pontivy), E. Ferrand (CH Cr\u00e9teil), A. Garioud (CH Creil), F. Skinazi (CH Saint-Denis), D. Bardou (CH Cholet), F. Heluwaert (CH Annecy), R. Arotcarena (CH Pau), M. Schnee (CH La Roche sur Yon), J.G. Bertolino (CH Gap), J.P. Dupuychaffray (CH Angoul\u00e8me), V. Crombe (CH Roubaix), A.M. Roque-Afonso (H\u00f4pital Paul Brousse, Villejuif) ; ANGH<\/em><\/p>\n<p><strong>H\u00e9patologie <\/strong>&#8211; \u00a02016-05-06 &#8211;\u00a0CO &#8211;<\/p>\n<p>________________________________<\/p>\n<p>Le virus de l\u2019h\u00e9patite E (VHE) pourrait \u00eatre l\u2019une des causes de d\u00e9compensation h\u00e9patique des malades cirrhotiques. Ainsi, une pr\u00e9valence de plus de 6% d\u2019infections VHE aigu\u00ebs a \u00e9t\u00e9 retrouv\u00e9e dans une \u00e9tude prospective et monocentrique Fran\u00e7aise r\u00e9alis\u00e9e chez des malades hospitalis\u00e9s pour d\u00e9compensation cirrhotique, le plus souvent d\u2019origine alcoolique.  De m\u00eame, 2 \u00e9tudes r\u00e9trospectives ont mis en \u00e9vidence une infection aigu\u00eb chez 3,6 \u00e0 6,5% des malades avec une h\u00e9patite alcoolique aigu\u00eb (HAA) s\u00e9v\u00e8re.  Toutefois, une \u00e9tude cas-t\u00e9moin, ancillaire d\u2019HALT-C, ne retrouvait pas le VHE, comme facteur de d\u00e9compensation h\u00e9patique chez des malades porteurs d\u2019une cirrhose virale C.<br \/>\nLe but de cette \u00e9tude \u00e9tait d\u2019\u00e9valuer la pr\u00e9valence du VHE dans une population de malades alcooliques pr\u00e9sentant une cirrhose d\u00e9compens\u00e9e associ\u00e9e ou non \u00e0 une h\u00e9patite alcoolique aigu\u00eb (HAA).<br \/>\nPatients et m\u00e9thodes\u00a0: les malades hospitalis\u00e9s pour d\u00e9compensation d\u2019une cirrhose alcoolique \u00e9taient inclus de mani\u00e8re cons\u00e9cutive dans 24 centres participants entre mars et juin 2015. Les malades b\u00e9n\u00e9ficiaient, le jour de l\u2019inclusion, d\u2019un interrogatoire exhaustif \u00e0 la recherche des diff\u00e9rents facteurs de risque d\u2019infection par le VHE. Le bilan biologique \u00e0 l\u2019inclusion comportait une s\u00e9rologie VHE (IgG et IgM, test Wantai) ainsi une recherche de l\u2019ARN viral dans le sang (test Ceeram).<br \/>\nR\u00e9sultats\u00a0: 181 malades dont l\u2019\u00e2ge moyen \u00e9tait de 58 ans ont \u00e9t\u00e9 inclus (sexe masculin=81%). Plus de la moiti\u00e9 d\u2019entre eux pr\u00e9sentaient un ant\u00e9c\u00e9dent de d\u00e9compensation h\u00e9patique, le plus souvent selon le mode ascitique. La majorit\u00e9 des malades \u00e9tait C au score de Child (A= 12%, B=57%, C=61%)\u00a0; le score moyen de MELD \u00e9tait de 18+\/-5,5. A l\u2019inclusion, 75% pr\u00e9sentaient une ascite, 62% un ict\u00e8re, 28% une enc\u00e9phalopathie h\u00e9patique, 21% une h\u00e9morragie digestive et 21% une infection. 61% des malades (81\/181) pr\u00e9sentaient un tableau clinico-biologique compatible avec une h\u00e9patite alcoolique aigu\u00eb (HAA), s\u00e9v\u00e8re chez 61% d\u2019entre eux (50\/81). Tous les malades avec une HAA s\u00e9v\u00e8re ont \u00e9t\u00e9 trait\u00e9s par cortico\u00efdes. La s\u00e9ropr\u00e9valence des IgG VHE \u00e9tait de 31%, non corr\u00e9l\u00e9e \u00e0 l\u2019\u00e2ge (p=\u00a00,40) ni au lieu de r\u00e9sidence (nord vs sud, 58 vs 42%, p=0,31). Des IgM VHE faiblement r\u00e9actifs ont \u00e9t\u00e9 retrouv\u00e9s dans 3 cas, avec un ARN VHE ind\u00e9tectable chez l\u2019ensemble des patients. La s\u00e9ropr\u00e9valence chez les sujets ayant pr\u00e9sent\u00e9 au moins un \u00e9pisode de d\u00e9compensation ant\u00e9rieure (57%) n\u2019\u00e9tait pas diff\u00e9rente de celle des sujets sans ant\u00e9c\u00e9dent de d\u00e9compensation (43%, p=0,78). Apr\u00e8s inclusion, le taux de d\u00e9c\u00e8s \u00e9tait de 6,5% \u00e0 14 jours, 14% \u00e0 30 jours et \u00e0 22% \u00e0 90 jours.<br \/>\nConclusion\u00a0: dans cette cohorte prospective, la d\u00e9compensation de la maladie alcoolique n\u2019\u00e9tait pas associ\u00e9e \u00e0 une infection aigu\u00eb par le VHE. Une s\u00e9ropr\u00e9valence \u00e9lev\u00e9e mais comparable \u00e9tait retrouv\u00e9e chez les malades avec ou sans ant\u00e9c\u00e9dent de d\u00e9compensation cirrhotique. Ainsi, malgr\u00e9 une pr\u00e9valence \u00e9lev\u00e9e de l\u2019infection du VHE dans la population de malades porteurs d\u2019une maladie alcoolique d\u00e9compens\u00e9e, aucun lien entre VHE et d\u00e9compensation h\u00e9patique n\u2019a \u00e9t\u00e9 retrouv\u00e9 dans cette \u00e9tude.<\/p>\n","protected":false},"excerpt":{"rendered":"<p>2016 C. Renou (CH Hy\u00e8res), B. Lesgourgues (CH Montfermeil), H. Agostini (H\u00f4pital Le Kremlin Bic\u00eatre, Le Kremlin Bic\u00eatre), G. Macaigne (CH Marne la Vall\u00e9e), A. Pauwels (CH Gonesse), Y. Le Bricquir (CH B\u00e9ziers), J. Henrion (CH Jolimont, Belgique), F. Khemissa (CH Perpignan), E. Clair (CH Montfermeil), T. Paupard (CH Dunkerque), A. Pelaquier (CH Mont\u00e9limar), H. [&hellip;]<\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_exactmetrics_skip_tracking":false,"_exactmetrics_sitenote_active":false,"_exactmetrics_sitenote_note":"","_exactmetrics_sitenote_category":0,"footnotes":""},"categories":[3],"tags":[27],"class_list":["post-2054","post","type-post","status-publish","format-standard","hentry","category-hepatologie","tag-27"],"yoast_head":"<!-- This site is optimized with the Yoast SEO plugin v27.2 - https:\/\/yoast.com\/product\/yoast-seo-wordpress\/ -->\n<title>R\u00f4le du virus de l\u2019h\u00e9patite E dans la d\u00e9compensation de la cirrhose alcoolique\u00a0? 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